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Table 1 Summary of clinical and laboratory data for cattle infected with piroplasms and Anaplasma marginale / A. ovis

From: Re-emergence of bovine piroplasmosis in Hungary: has the etiological role of Babesia divergens been taken over by B. major and Theileria buffeli?

Animal no. Month when diseased (tested) Age in year Main clinical signs Laboratory findings Status/outcome
     Haematocrit Blood smear Molecular  
1. November 3 lethargy, ataxia, oedema, recumbency1 27% Bm ne death in 2 wk
2. December 3 lethargy, oedema, recumbency ne Bm ne slaughtered
3. December 10 haemoglobinuria, recumbency ne Bm ne death in 4 wk
4. (January) 0.3 - 30% ne Bm healthy
5. (January) 0.5 - normal ne Bm + Am healthy
6. (January) 8 anaemia 22% ne Tb treated: recovery
7-9. (January) 5,7,10 - normal ne Tb + Am healthy
  1. 1Clinical laboratory findings of animal No 1. [normal range]:
  2. - elevated: aspartate-aminotransferase (AST): 337.2 [10–160] IU/l, alkaline phosphatase (AP): 310 [40–200] IU/l, glutamate dehydrogenase (GLDH): 116.8 [1025] U/l, carbamid: 6.6 [1–3.7] mmol/l, lactate dehydrogenase (LDH): 10050 [<450] U/l.
  3. - lowered: albumin: 23.7 g/l [35–42].
  4. Abbreviations: Bm Babesia major, Tb Theileria buffeli, Am Anaplasma marginale/A. ovis, ne not evaluated, wk week(s).