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Table 1 Summary of clinical and laboratory data for cattle infected with piroplasms and Anaplasma marginale / A. ovis

From: Re-emergence of bovine piroplasmosis in Hungary: has the etiological role of Babesia divergens been taken over by B. major and Theileria buffeli?

Animal no.

Month when diseased (tested)

Age in year

Main clinical signs

Laboratory findings

Status/outcome

    

Haematocrit

Blood smear

Molecular

 

1.

November

3

lethargy, ataxia, oedema, recumbency1

27%

Bm

ne

death in 2 wk

2.

December

3

lethargy, oedema, recumbency

ne

Bm

ne

slaughtered

3.

December

10

haemoglobinuria, recumbency

ne

Bm

ne

death in 4 wk

4.

(January)

0.3

-

30%

ne

Bm

healthy

5.

(January)

0.5

-

normal

ne

Bm + Am

healthy

6.

(January)

8

anaemia

22%

ne

Tb

treated: recovery

7-9.

(January)

5,7,10

-

normal

ne

Tb + Am

healthy

  1. 1Clinical laboratory findings of animal No 1. [normal range]:
  2. - elevated: aspartate-aminotransferase (AST): 337.2 [10–160] IU/l, alkaline phosphatase (AP): 310 [40–200] IU/l, glutamate dehydrogenase (GLDH): 116.8 [1025] U/l, carbamid: 6.6 [1–3.7] mmol/l, lactate dehydrogenase (LDH): 10050 [<450] U/l.
  3. - lowered: albumin: 23.7 g/l [35–42].
  4. Abbreviations: Bm Babesia major, Tb Theileria buffeli, Am Anaplasma marginale/A. ovis, ne not evaluated, wk week(s).